Sounds like typical Kennedy carp to me. Funny that she reported Ambien as something she probably took. Again who the heck takes Ambien in the MORNING!!!! That is NOT a drug you should drive on. But she isn't the only Kennedy who did that.
Seems like alcohol, drugs, refusing to take responsibility for their actions and lying are Kennedy family traditions. They truly feel they are above the law and that shouldn't be held accountable for their wrongdoings.
Now if she really did have a seizure then she should take note of the following laws about driving and seizures in New York
•You must be seizure free for one year.
• A person is disqualified from driving a commercial motor vehicle if that person has a medical history of epilepsy, has a current clinical diagnosis of epilepsy or is taking antiseizure medication.
• Doctors are not required to report epilepsy.
• Exceptions may be granted by the DMV’s Medical Review Board. Periodic medical updates are required after licensing if determined by the DMV.
• DMV appeal of license denial must be filed within 30 days
Now of course, being a Kennedy, these laws won't apply to her. No doubt there will be a special loophole that allows her to keep driving.
I will say that I did go thru a period in my life when I suffered with mysterious passing out, seizure like episodes. I voluntarily gave up driving during that time and haven't had one in almost 4 years. Luckily my late fiance was a first responder so he had no problems dealing with them and he could sometimes even predict them and tell me to go lay down.
But I seriously doubt she had a seizure. Just sounds like more Kennedy CYA carp. The fact that they have a family historian is amusing to say the least. maybe if they would just tell the truth, they wouldn't have to have someone to rewrite their history.
I never heard of the drug Ambien.
Zolpidem (sold under the brand names Ambien, Ambien CR, Stilnox, and Sublinox) is a prescription medication used for the treatment of insomnia, as well as some brain disorders.
Residual 'hangover' effects, such as sleepiness and impaired psychomotor and cognitive function, after nighttime administration may persist into the next day, which may impair the ability of users to drive safely and increase risks of falls and hip fractures.
It is a short-acting nonbenzodiazepine hypnotic of the imidazopyridine class [2] that potentiates gamma-aminobutyric acid (GABA), an inhibitory neurotransmitter, by binding to GABAA receptors at the same location as benzodiazepines.[3] It works quickly (usually within 15minutes) and has a short half-life (two to three hours).
Zolpidem has not adequately demonstrated effectiveness in maintaining sleep (unless delivered in a controlled-release form); however, it is effective in initiating sleep.[4] Its hypnotic effects are similar to those of the benzodiazepine class of drugs.
As an anticonvulsant and muscle relaxant, the drug's effects aren't evident until dosages 10 and 20 times those required for sedation, respectively, are reached. For that reason,
zolpidem has never been approved for either muscle relaxation or seizure prevention. Such drastically increased doses are also more inclined to induce one or more of the drug's adverse side effects, including hallucinations and amnesia.
There is evidence that zolpidem can rouse patients from a persistent vegetative state.
Zolpidem 10 mg is effective in treating insomnia when used intermittently no fewer than three and no more than five pills per week for a period of 12 weeks.[28] The 15-mg zolpidem dosage provided no clinical advantage over the 10-mg zolpidem dosage.
Alcohol has cross tolerance with GABAA receptor positive modulators such as the benzodiazepines and the nonbenzodiazepine drugs. For this reason, alcoholics or recovering alcoholics may be at increased risk of physical dependency on zolpidem. Also, alcoholics and drug abusers may be at increased risk of abusing and or becoming psychologically dependent on zolpidem. It should be avoided in those with a history of alcoholism, drug misuse, physical dependency, or psychological dependency on sedative-hypnotic drugs. Zolpidem has rarely been associated with drug-seeking behavior, the risk of which is amplified in patients with a history of drug or alcohol abuse.
Detection in body fluids
Zolpidem may be quantitated in blood or plasma to confirm a diagnosis of poisoning in hospitalized patients, provide evidence in an impaired driving arrest, or to assist in a medicolegal death investigation. Blood or plasma zolpidem concentrations are usually in a range of 30–300 μg/l in persons receiving the drug therapeutically, 100–700 μg/l in those arrested for impaired driving, and 1000–7000 μg/l in victims of acute overdosage. Analytical techniques, in general, involve gas or liquid chromatography.
Perpetrators of sexual assault have used zolpidem on unsuspecting victims.
Special precautions: Driving
Use of zolpidem may impair driving skills with a resultant increased risk of road traffic accidents. This adverse effect is not unique to zolpidem but also occurs with other hypnotic drugs. Caution should be exercised by motor vehicle drivers.
Adverse effects•
• Vomiting & Nausea
• Dizziness
• Anterograde amnesia
• Hallucinations, through all physical senses, of varying intensity[citation needed]
• Delusions[citation needed]
• Altered thought patterns
• Ataxia or poor motor coordination, difficulty maintaining balance[17]
• Euphoria and/or dysphoria
• Increased appetite
• Increased or decreased libido
• Amnesia
• Impaired judgment and reasoning
• Uninhibited extroversion in social or interpersonal settings
• Increased impulsivity
• When stopped, rebound insomnia may occur
• Headaches
• Short-term memory loss
http://en.wikipedia.org/wiki/Ambien
